2015 Annual Show - Information on our Judge

Name: Patrick Dooner

Patrick comes from a long, showing and judging career...

He is a registered IKC judge for Terrier Group 3  and has Judged in Ireland as well as Europe.

He has won several top prices showing in Crufts with his parson Russell’s. His kennel name is 'Jappatin Parson Russell'. Patrick is a long time admirer of glens and is really pleased to be asked to Judge this year’s Sporting club show...

We are looking forward to seeing Patrick on the day!

Interesting Doggie Facts

I came upon these two websites recently, and thought they were very good. They have various facts on  'man's best friend'...  Click here for funny dog facts. Click here to see Sybils Den info. You might be surprised by some of them!


Message from Mr. Pascal Tyndall, Judge for 2012 Annual Show

"My name is Pascal Tyndall from Dordrecht, the Netherlands.

Annelies van Wijk & I got our first Irish Glen of Imaal Terrier, LouLou of Megh-Morran, in 1988. 20 years ago we had our first litter and we kept Phoebe Ailis Tailwaggor Tyndall. With Phoebe I flew to Lisboa (Portugal) and she became European Ch. Her mother, LouLou, got this title in Budapest (Hungary) in 1993. We have bred 7 litters. All of our own dogs were Multi Ch.
We exported 2 puppy's to Denmark & 1 to Norway & 1 to the USA.

At the moment, we have 1 Irish Glen of Imaal Terrier, called Gaia Gaia Tailwaggor Tyndall, she is eleven years old. Her sister, Kelsey, got the title World Winner in 2003.

In 1995, I initiated to start a own breed club for the Glen. I became Secretary of this club until 2009. Since April 2009 I'm president of the Irish Glen of Imaal Terrier Club Nederland. In 2000, I was invited by Eithne Cleary to give a lecture on the Irish Glen of Imaal Terrier in Dublin, which I did off course!

My first experience with judging our breed was in Germany at the Irish Native Terrier Breed Festival in 2009. 34 Glens had entered, a record for Germany.

When judging the Irish Glen of Imaal Terrier it's important for me: type, head, earcarriage & placement, good filled up forface, powerfull neck, placement of shoulders, length of upperarm, ribcage, angulation of hindquarters & very import effortless moving!

Hope to see you on September 9th!"

Pascal Tyndall

Photo Competition

Dont forget to email your favourite Glen photo to our committee for entry into this years photo competition. The best photo will be used on the coverpage of our newsletter.

If you would like further information, contact our committee members on sportingglens@yahoo.ie

What is PRA?

PRA (Progressive Retinal Atrophy) refers to a group of diseases that cause the retina of the eye to degenerate slowly over time. The result is declining vision and eventual blindness. A company called "OptiGen" now provides a new DNA test that detects the mutation causing an inherited eye disease, Cone Rod Dystrophy 3 (crd3), in the Glen of Imaal Terrier.

PRA Disease

The genetic disorder,crd3 is one of several cone rod dystrophies that have been recognized in multiple breeds of dogs. All forms of CRD are characterized by the initial loss of cones, the cells in the retina that are responsible for vision in bright light/daylight, followed by the degeneration of rods, the retinal cells that operate during night vision.

Clinical symptoms of crd3 are usually not evident until adulthood has been reached. In some ways, the clinical appearance of crd3 is similar to other common forms of late-onset PRA, namely there is retinal thinning that can be observed by a veterinary ophthalmologist in dogs as young as 3 years of age and retinal degeneration progression, resulting in total blindness within a few years. The Glen of Imaal Terrier’s small population size and its limited gene pool, along with the adult onset of disease -- often after an animal has been bred multiple times-- have resulted in widespread distribution of crd3 within the breed, and many Glens in the general population are likely to be carrying at least one copy of the disease mutation.

Unfortunately, at this time there is no treatment or cure for PRA.


PRA is inherited as a recessive trait. This means a disease gene must be inherited from each parent in order to cause disease in an offspring. Parents were either “carriers” or "affected".

A normal/clear dog has no disease gene and is termed “homozygous normal” – both copies of the gene are the same.

A carrier has one disease gene and one normal gene, and is termed “heterozygous” for the disease.

And an affected dog with two disease genes is termed “homozygous affected” – both copies of the gene are abnormal.

Although PRA is inherited, it can be avoided in future generations by testing dogs before breeding. Identification of dogs that do not carry disease genes is the key. These "clear" dogs can be bred to any mate - even to a crd3-affected dog which may be a desirable breeding prospect for other reasons. The chance of producing affected pups from such breedings depends on the certainty of test results.

The Genetic Test

The OptiGen crd3 test is done on a small sample of blood from the dog. The test analyzes the specific DNA mutation causing the PRA, crd3 strain. The OptiGen test detects the mutant, abnormal gene copy and the normal gene copy.

The result of the test is a genotype and allows separation of dogs into three groups:

  • Normal/Clear (homozygous normal)
  • Carrier (heterozygous)
  • Affected (homozygous affected)
Possible PRA Breeding Results
Parents Result Risk of PRA Disease
Normal/Clear Can be bred to any dog, extremely low risk of producing affected offspring
Carrier Should be bred to Normal/Clear to reduce risk of producing affected offspring
Affected Should be bred to Normal/Clear to reduce risk of producing affected offspring
For more detailed information, please go to the following website: Optigen